How to stop Alzheimer’s

8 Ways to Prevent Alzheimer’s Disease

Keep your brain healthy, sharp, and reduce the risk of Alzheimer’s with these scientifically-proven strategies

1. Eat More Fruits and Vegetables

A population-based cohort study of 1,836 older Japanese-Americans found that consumption of fruit and vegetable juices was associated with decreased incidence of Alzheimer’s over seven to nine years of follow-up.

3. Reach for Berries
Berries contain high levels of biologically active components, including a class of compounds called anthocyanosides, which fight memory impairment associated with free radicals and beta-amyloid plaques in the brain. Eat berries each day for maximum benefit.

3. Increase Omega-3 Fatty Acids

In the Framingham study, individuals with the top quartile levels of docosahexaenoic acid (DHA) found in fatty fish like salmon, mackerel, and tuna, measured at baseline had lower rates of Alzheimer’s over nine years of follow-up. These fish are all rich in Omega-3 fatty acids.

4. Take Folic Acid Supplements

If you don’t take a supplement, eat foods high in folate. High levels of homocysteine may be associated with poor cognitive function. Some findings indicate that reducing homocysteine with folic acid may increase cognitive function.

5. Sip Smarter

Drink a glass of red wine or purple grape juice with your evening meal. Components in grape skins protect brain cells from the toxic effect of oxidative stress and beta amyloid.

6. Go Mediterranean

Two studies that used dietary questionnaires to assess and quantify adherence to the diet in different populations found that patients who were most adherent to the Mediterranean style diet had a lower incidence of Alzheimer’s, compared with those who did not follow this diet.

7. Control Your Blood Pressure

Hypertension appears to be associated with an increased risk of both vascular dementia and Alzheimer’s disease.

8. Have Strong Social Support

Findings indicate that an active social life and strong network of friends may help prevent Alzheimer’s in later life.

Buy the book and learn more about The Anti-Alzheimer’s Prescription


$200 billion will be paid for Alzheimer’s care

In 2012, the direct costs of caring for those with Alzheimer’s or other dementias to American society will total an estimated $200 billion.Average per person Medicare payments for an older person with Alzheimer’s or other dementias are nearly 3 times higher than for an older person without these conditions. Medicaid payments are 19 times higher. These costs will only continue to soar in the coming years given the projected rapidly escalating prevalence of Alzheimer’s disease as the baby boomers age.
Unless something is done, the care costs of Alzheimer’s and other dementias will soar from $200 billion this year to a projected $1.1 trillion (in today’s dollars) by 2050. This dramatic rise includes a 500 percent increase in combined Medicare and Medicaid spending.


Wednesday, December 19, 2012

The Starving Brain cause of Alzheimer’s

A reduction in cerebral glucose utilization is one of the earliest signs of
Alzheimer’s disease.
Although the exact cause of this reduction is not known,
gathering evidence suggests that it is part of a complex metabolic adaptation to
oxidative stress during which glycolysis and oxidative phosphorylation are turned
down, glucose metabolism is shifted to the pentose phosphate pathway
to generate antioxidant reducing factors such as nicotinamide adenine dinucleotide
phosphate (NADPH) and glutathione, and the gamma-aminobutyric acid (GABA)
shunt is activated to provide glutamate as an alternate source of energy.In the face of these adaptive metabolic changes, the Alzheimer brain runs short
of energy and begins to digest itself.

Evidence is presented that gammahydroxybutyrate, a natural product of the GABA
shunt, can provide the necessary energy, carbon, and antioxidant power and that
its use may be able to delay the onset and progress of Alzheimer’s disease. ”


Thursday, November 29, 2012

Fat Will Eat Your Mind

It is already known that both excess visceral fat and the lifestyle choices needed to gain it – being sedentary and a high calorie diet – correlate with increased risk of many age-related conditions. Going beyond associations to matters of causation, it is worth noting that animal studies have shown that surgical removal of visceral fat increases life expectancy.
These recently published open access study results add to the existing stack of reasons to care about your weight and lifestyle choices:
The purpose of this study was to determine the association between visceral adipose tissue (VAT) and all-cause mortality. The sample included 1089 white men and women 18-84 years of age from the Pennington Center Longitudinal Study, a prospective cohort of participants assessed between 1995 and 2008, and followed for mortality until 31 December 2009. Abdominal VAT was measured [using] computed tomography. There were 27 deaths during an average of 9.1 years of follow-up. Source:

Saturday, November 24, 2012

Niacin intake protect against the development or progression of Alzheimer’s disease

Nicotinamide restores cognition in AD transgenic mice via a mechanism involving sirtuin inhibition
and selective reduction of Thr231-phosphotau.Source:http://www.ncbi.nlm….cles/PMC2617713

Cell Life Versus Cell Longevity: The Mysteries Surrounding the NAD+ Precursor Nicotinamide.
” In a similar vein, inhibition of PARP activity by nicotinamide may be critical for disorders such as Alzheimer’s disease.

More recently, sufficient dietary niacin intake examined in a series of patients aged 65 and older has been implicated as a potential factor to protect against the development or
progression of Alzheimer’s disease. ”


Choline + Uridine: Build New Neurons and Protect Against Alzheimer’s

” Choline + Uridine: Build New Neurons and Protect Against Alzheimer’s ”

Aging influences the
micro environment for adult and immature neurons in the brain, which may affect the proliferation and migration of neural stem/progenitor cells, and YKS has pharmacological potency for these  age-related events. These findings help to understand the physiology and pathology of the aged brain and provide an anti-aging strategy for the brain. ”


” YKS has improving activity for age-related increased anxiety and enhances serotonergic and dopaminergic transmissions in the aged PFC. These mechanisms provide information important for the treatment of anxiety in the elderly. ”


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