Averting a Pandemic? Scientists Re-engineer 1918 Spanish Flu

May 1st, 2014

FAKE NEWS for the Zionist agenda

Orig.src.Susanne.Posel.Daily.News- spanish.flu.bioweapon.university.arizona.pandemic_occupycorporatism Susanne Posel ,Chief Editor Occupy Corporatism | The US Independent
April 30, 2014

Researchers at the University of Arizona (UA) have been studying the 1918 influenza virus that killed an estimated 50 million people to decipher vaccination strategies in the name of avoiding another pandemic.

According to historian Alfred Crosby, this virus killed “more people in a year than the bubonic plague killed in a century in the Middle Ages.”

This particular virus has prompted the development of a “molecular clock” to analyze “the rate at which mutations build up in specific parts of DNA.”

The researchers “reconstructed” this virus with the swine and seasonal flus to understand how the virus was able to infect so many to become so deadly.

The mode of thinking was: “It sounds like a modest little detail, but it may be the missing piece of the puzzle. Once you have that clue, many other lines of evidence that have been around since 1918 fall into place.”

From previous work, it has been determined that the 1918 influenza originated from a human H1 and somehow “picked up” genetic material from an avian flu to create the new virus that began circulating in the early 1900s.

Interestingly, the human immune system, when exposed to a virus, will build up anti-bodies to better fight off the virus if ever the body comes into contact with it.

Researchers concluded that those who survived the 1918 pandemic must have been exposed to H3N8 which is believed to be in circulation at the time.

Michael Worobey, lead author of the study, explained : “Ever since the great flu pandemic of 1918, it has been a mystery where that virus came from and why it was so severe, and in particular, why it killed young adults in the prime of life. It has been a huge question what the ingredients for that calamity were, and whether we should expect the same thing to happen tomorrow, or whether there was something special about that situation.”

Worobey said that his team discovered: “A person with an antibody arsenal directed against the H3 protein would not have fared well when faced with flu viruses studded with H1 protein. And we believe that that mismatch may have resulted in the heightened mortality in the age group that happened to be in their late 20s during the 1918 pandemic.”

With childhood exposure, the researchers believe that protection from the fatal effects of the 1918 influenza could be averted in the future.

Worobey asserted: “The new perspective does not just apply to the pandemic of 1918, but might also explain patterns of seasonal flu mortality and the mysterious patterns of mortality from highly pathogenic avian origin H5N1. H5N1 causes higher mortality rates in young people and H7N9 causes higher mortality in the elderly. In both cases, the more susceptible age groups were exposed initially, as children, to viruses with a mismatched HA, and may suffer severe consequences similar to young adults faced with a mismatched virus in 1918.”