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June 14th, 2012 
FAKE NEWS for the Zionist agenda THURSDAY, June 14 (HealthDay News) — Pregnant women’s immune
systems do not attack their developing babies because embryo implantation
in the uterus triggers a process that affects the ability of immune cells
to reject foreign bodies, new research shows.
The investigators from the NYU Langone Medical Center found a key
immune system pathway is turned off following implantation, so immune
cells are not called in to harm a fetus. Without this process, the
researchers noted, preterm labor, miscarriage or a dangerous medical
condition called preeclampsia could result.
The study was published recently in the journal Science.
According to lead investigator Dr. Adrian Erlebacher, an associate
professor of pathology at the medical center, the study “addresses a
fundamental question . . . namely, how do the fetus and placenta, which
express antigens that are disparate from the mother, avoid being rejected
by the maternal immune system during pregnancy?” The answer, he explained
in a center news release, “was completely unexpected at every level.”
Normally, the immune system produces chemokines, which trigger immune
cells to attack foreign tissues, such as occurs in the typical tissue
rejection response that occurs following an organ transplant.
During pregnancy, however, women’s immune cells come into contact with
the foreign antigens of their developing fetus and placenta, but this
rejection response does not take place. To investigate why this happens,
the researchers examined the decidua, the structure that contains the
fetus and placenta.
The findings showed that when a woman becomes pregnant, the genes
responsible for calling immune cells are turned off inside the decidua,
protecting the developing fetus. The study authors explained that an
“epigenetic change,” or a non-hereditary change, takes place in the DNA of
the cells of the decidua that deactivates the chemokine genes. When this
happens, the typical immune system response is deactivated at the site of
embryo implantation.
“It turns out that the cells that typically secrete the
chemoattractants to bring the T-cells [the cells that accumulate and
attack tissue] to sites of inflammation are inhibited from doing so in the
context of the pregnant uterus,” Erlebacher said. “The decidua appears
instead as a zone of relative immunological inactivity.”
In addition to pregnancy, the study authors said their findings could
also have implications for autoimmune diseases, organ transplantation and
cancer.
“This is a very exciting finding for us because it gives a satisfying
explanation for why the fetus isn’t rejected during pregnancy, which is a
fundamental question for the medical community with clear implications for
human pregnancy,” Erlebacher said. And, he added, it could lead to new
methods for treating many other conditions and diseases.
More information
The U.S. National Library of Medicine has more about the immune system.
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