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February 4th, 2016 
Awake Goy 
Susanne Posel ,Chief Editor Occupy Corporatism | Co-Founder, Legacy Bio-Naturals
Mayo Clinic researchers say they have found the secret to the fountain of youth – or at least a possible way of extending life by irradiating a specific cell that accumulates during the course of a human lifetime.
Affectionately called senescent cells, these are normal cells that have ceased to divide due to “unusual amounts of stress” and simply “decided to stop dividing”.
It may come as no surprise that older animals have more senescent cells than their younger counterparts. And while it appears these cells are dormant, they actually secrete an inflammation causing chemical cocktail that happens to be present “in just about every major age-related disease.”
In this study, the researchers genetically engineered mice that would respond to a “drug [that] would trigger senescent cells to kill themselves” and discovered that as “the mice got older” they appeared to be “healthier”.
These mice also lived 25% longer than their untreated counterparts simply by having “their senescent cells removed from 1 year of age on”.
In addition, the mice succumbed to less aging-related diseases such as:
• Cataracts
• Stress related heart problems
• Kidney malfunction
The researchers concluded that “it seems like we’re accumulating a cell type that we really don’t need for anything and that makes us more unhealthy and reduces the length of our healthy lives.”
This new study compliments a previous publication by Stanford University (SU) Baxter Laboratory for Stem Cell Biology (BLSCB) who found a viable way of producing anti-aging properties in cells.
By modifying RNA, the researchers created “large numbers of cells for study or drug development.”
Skin cells with telomeres lengthened by the procedure were able to divide up to 40 more times than untreated cells. In essence, this technique could be used to stop aging which is caused by the shortening of telomeres over a lifetime.
Telomeres are the “are the caps at the end of each strand of DNA that protect our chromosomes, like the plastic tips at the end of shoelaces.”
New telomeres are 8,000-10,000 nucleotides long and shorten as cells divide. When they reach their maximum division, cells die which is the internal aging clock for humans and other life.
Helen Blau, professor of microbiology and immuniology at SU explained: “Now we have found a way to lengthen human telomeres by as much as 1,000 nucleotides, turning back the internal clock in these cells by the equivalent of many years of human life. This greatly increases the number of cells available for studies such as drug testing or disease modeling.”
The injection of RNA to extend telomeres’ was successful.
RNA which “contained the coding sequence for TERT, the active component of a naturally occurring enzyme called telomerase. Telomerase is expressed by stem cells, including those that give rise to sperm and egg cells, to ensure that the telomeres of these cells stay in tip-top shape for the next generation. Most other types of cells, however, express very low levels of telomerase.”
Researchers proposed that “the development of telomere extension [could] improve cell therapies and to possibly treat disorders of accelerated aging in humans.”
Implications for diseases such as cardiovascular, diabetes, “conditions of aging” are now potentially in the realm of treatable with this discovery.
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