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April 11th, 2012 
FAKE NEWS for the Zionist agenda WEDNESDAY, April 11 (HealthDay News) — An experimental drug
reversed many of the major symptoms of Fragile X syndrome in mice,
researchers report.
Fragile X syndrome is a leading cause of autism, and its symptoms
include developmental delays, hyperactivity, learning and memory
difficulties, low IQ, seizures and social and communication deficits.
Prior research suggested that inhibiting mGlu5, a receptor for a
neurotransmitter known as glutamate, might help alleviate some
symptoms.
In the new study, researchers in Switzerland and at the Massachusetts
Institute of Technology treated adult mice genetically engineered to have
Fragile X syndrome with a compound called CTEP, which inhibits mGlu5.
“We found that even when treatment with CTEP was started in adult mice,
it reduced a wide range of [Fragile X] symptoms,” study author Dr. Lothar
Lindemann, of F. Hoffmann-La Roche Ltd. in Basel, said in a journal news
release.
Symptoms that showed improvement included learning and memory
challenges and hypersensitivity to sounds.
Although studies involving animals can be useful, they frequently fail
to produce similar results in humans, experts noted. The study’s authors
also pointed out that CTEP was not developed for use in people. The
findings, however, could help scientists pursue a similar treatment for
people with Fragile X.
“The most important implications of our study are that many aspects of
[Fragile X] are not caused by an irreversible disruption of brain
development, and that correction of the altered glutamate signaling can
provide widespread therapeutic benefit,” said Mark Bear, of MIT’s Picower
Institute for Learning and Memory.
The study is published in the April 12 issue of the journal
Neuron.
More information
The U.S. National Institutes of Health provides more information on Fragile X syndrome.
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